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. 2017 Nov 1;37(44):10690–10699. doi: 10.1523/JNEUROSCI.2141-17.2017

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Copyright © 2017 the authors 0270-6474/17/3710690-10$15.00/0

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Figure 5. — CaMKII-mediated phosphorylation of NMDARs in PVNs in WKY rats and SHRs. A, B, Representative gel images (A) and summary data (B) show the phosphorylated GluN2B (Ser1303) levels (normalized to GAPDH) in the PVN, hippocampus, and frontal cortex in WKY rats and SHRs (n = 6 rats in each group). C, Original ABP traces and group data show the effect of CGx and sham surgery on the mean ABP in SHRs (n = 6 rats in each group). D, Representative images and summary data show the phosphorylated GluN2B (Ser1303) levels (normalized to GAPDH) in the PVN in SHRs subjected to CGx or sham surgery (n = 6 rats in each group). E, Summary data show the mRNA levels of CaMKII-α and CaMKII-β subunits in PVN tissues from WKY and SHR (n = 7 rats in each group). F, Representative gel images and summary data show that CaMKII-α and CaMKII-β protein levels in the PVN tissue in WKY rats and SHRs (n = 6 rats in each group). Data are presented as the mean ± SEM. *p < 0.05 compared with the WKY group; #p < 0.05 compared with the sham group.