Figure EV1. OHC electromotility remains unaffected in Lrba mutants.

Prestin‐driven electromotility remains functional in Lrba‐KO OHCs (p15–16).

• A
  Motor protein levels in the OHC membrane leaflet were determined electrophysiologically by analyzing the nonlinear capacitance (NLC) of WT and Lrba‐KO OHCs in response to a stair‐step protocol.
• B, C
  Representative current traces to quantify; (B) NLC voltage dependence; and (C) peak capacitance. In these latter experiments, no statistically significant differences between WT and Lrba mutants could be detected (WT n = 10, Lrba‐KO n = 9; P = 0.063; Student's t‐test). Data are presented as means ± s.e.m.