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. 2017 Sep 1;14(5):5285–5292. doi: 10.3892/ol.2017.6874

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Copyright: © Arnold et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Graphical representation of the number of patients who (A) exhibited recurrence or (B) who had survived at the completion of this study in comparison to Hedgehog (HH) or Wnt activation. Patients were divided into three groups: No activation of HH or Wnt pathways, only one pathway activated or both pathways activated. Patients with dual activation of HH and canonical Wnt had poorer survival and were at greater risk of recurrence.

Figure 3. — Graphical representation of the number of patients who (A) exhibited recurrence or (B) who had survived at the completion of this study in comparison to Hedgehog (HH) or Wnt activation. Patients were divided into three groups: No activation of HH or Wnt pathways, only one pathway activated or both pathways activated. Patients with dual activation of HH and canonical Wnt had poorer survival and were at greater risk of recurrence.