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. 2017 Nov 2;14:23. doi: 10.1186/s12950-017-0170-7

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Comparison of TUNEL, hematoxylin and eosin (H&E), and myeloperoxidase (MPO) staining in control and experimental NHPs. a-b: TUNEL staining in control (a) and experimental NHPs (b) demonstrated a significant difference in pneumocyte apoptosis in the experimental, polytrauma group (g). c-d: H&E staining of control (c) and experimental NHPs (d) showing increased airspace edema and alveolar wall thickening in the experimental group. e-f: Comparison of MPO activity in control (e) versus experimental NHPs (f) revealed a significant difference accumulation of MPO⁺ activated neutrophils in the polytrauma arm (h)