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. 2017 Nov 2;8:1258. doi: 10.1038/s41467-017-01342-5

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — RNA-Seq-derived expression levels of γ AMPK isoforms and gene ontology analysis of iSABs vs aCaBs. a, b Prkag2 (a) and Prkag1 (b) gene expression in iSABs (induced sinoatrial bodies) vs aCaBs (antibiotic-selected cardiac bodies—a mixture of cardiomyocyte subtypes) by RNA-Seq (n = 3). c, d Functional annotation with gene ontology (GO) analysis of iSAB gene expression identifying enrichment of GO terms associated with pacemaking (c) and significant enrichment of AMPK-dependent downstream targets and ontological processes (d). a, b Data are shown as means ± s.e.m.