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. 2017 Nov 3;7:14597. doi: 10.1038/s41598-017-15058-5

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Response of arterial blood pressure (panel A), total renal blood flow (B), cortical laser flux (C), medullary laser flux (D), cortical tissue pO₂ (E), and medullary tissue pO₂ (F) to 45 min of unilateral warm renal ischemia (from time −45 min to time 0 min) followed by 60 min of reperfusion. Data are relative changes as related to the values recorded immediately before commencement of the renal ischemia (mean ± SEM; n = 9 for the saline group [control], n = 11 for the nitrite group); *denotes p < 0.05 control vs. nitrite group, significance bars indicate data that significantly differ from their pre-ischemia values, with x denoting p < 0.05 in the control group and +denoting p < 0.05 in the nitrite group.