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. 2017 Feb 15;32(6):1053–1061. doi: 10.3904/kjim.2016.060

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Copyright © 2017 The Korean Association of Internal Medicine

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted noncommercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — In vivo blockade of junctional adhesion molecule C (JAM-C) resulted in faster resolution of neutrophilic inflammation. (A) The number of infiltrated neutrophils in the kidney significantly decreased in the JAM-C blocking antibody (Ab)-treated group, compared to that found in the control immunoglobulin G (IgG)-treated group on day 5 after cisplatin injection (lymphocyte antigen 6 complex, locus G [Ly6G] stain, ×200; n = 6 animals per group). (B) The expression of interleukin 6 (IL-6) significantly decreased in the JAM-C blocking Ab-treated group (n = 6 animals per group). Tumor necrosis factor α (TNF-α) levels showed no statistically significant differences between groups. Cis, cisplatin; HPF, high-power field. ^ap < 0.05, compared with the sham or control IgG group.