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. 2017 Jul 26;8(47):82991–83008. doi: 10.18632/oncotarget.19635

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Copyright: © 2017 Liang et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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(A-C) miR-27a-3p antagomir significantly suppressed tumor growth (A), tumor weight (B), and tumor volume (C) of HCT116 cells implanted subcutaneously in BALB/c-nu mice compared with the control group, respectively. (D-F) miR-27a-3p agomir significantly increased tumor growth (D), tumor weight (E), and tumor volume (F) of SW480 cells implanted subcutaneously in BALB/c-nu mice compared with the control group, respectively. (G) Histological staining showed the lung metastatic carcinoma (arrow indicated) of tumor xenografts generated by HT29 cells transfected with miR-27a-3p agomir compared with the control group (NC), haematoxylin and eosin staining ×200. (H) miR-27-3p expression was significantly higher in tumor xenografts generated by HT29 cells transfected with miR-27a-3p agomir compared with the control group (NC) by quantitative real-time PCR.