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. 2017 Oct 30;11:343. doi: 10.3389/fncel.2017.00343

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Copyright © 2017 Hoxha, Gabriele, Balbo, Ravera, Masante, Zambelli, Albergo, Mitro, Caruso, Di Gregorio, Brusco, Borroni and Tempia.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Elovl5^−/− mice show cerebellar atrophy. (A–F) Nissl-stained sagittal sections of the cerebellum from wild type (A–C) and Elovl5^−/− mice (D–F) at 6, 12 and 18 months of age. (G–I) Average thickness of the molecular layer and (J–L) granular layer of each cerebellar lobule for Elovl5^−/− (red) and wild type mice (blue) at 6, 12 and 18 months, respectively. (M,O,Q) Reduced total perimeter length of the PC layer and (N,P,R) white matter area in the cerebellum of Elovl5^−/− mice at all ages analyzed. *p < 0.05; **p < 0.01, ***p < 0.001 two way ANOVA. Results are reported as mean ± SEM. Scale bar 100 μm.