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. 2017 Nov 3;8:1311. doi: 10.1038/s41467-017-00956-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Domain-specific modulation during thought and action suppression. a and b. Group (N = 24) whole-brain contrasts for No-Think < Think (top) and Stop < Go (bottom). Thought suppression modulated bilateral hippocampal (HIP) activity. Action-stopping-modulated activity in primary motor cortex (M1), lateralized to the left (contralateral to hand) hemisphere. Boxes illustrate HIP and M1 activations on a coronal slice in MNI space. Activations are derived from an uncorrected cluster-defining threshold (p < 0.001), with cluster level false discovery rate p < 0.05. Color bars demarcate T-statistics. (Middle panels) A priori region of interest (ROI) analyses: Group hemodynamic time-courses were attenuated in HIP by thought suppression (No-Think) and in M1 by action suppression (Stop) relative to Think and Go, respectively ***p < 0.001; **p < 0.01; *p < 0.05. c Modality-dependent hemodynamic attenuation in HIP (top) and M1 (bottom) was confirmed with a repeated measures ANOVA, which revealed an ROI by Modality interaction. Error bars represent SEM