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. 2017 Jul 20;80(5):895–907. doi: 10.1007/s00280-017-3392-8

Table 2.

Increased excretion
GI losses
 Diarrhea, laxative abuse, gastric suctioning
Renal losses
 Loop and thiazide diuretics
 Osmotic diuresis (uncontrolled diabetes)
 Post-obstruction
Hyperaldosteronism
 Primary hyperaldosteronism
 Corticosteroids
 Magnesium depletion
 Platinum agents
 Alcoholism
Cellular shifts
 Insulin administration
 β-Adrenergic agonists (bronchodilators, decongestants, theophylline)
 Acute catecholamine surge (e.g., myocardial infarction)