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Proposed model for histone deacetylation in memory retention. Model for learning and memory shows a role for Gug. At the start of memory formation, histone acetylases are upregulated following neuronal stimulation, promoting upregulation of transcription of memory related genes (A). Following memory consolidation, these gene sites on chromatin may become deacetylated, in addition to memory extinction genes, to promote memory retention (B).
