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. 2017 Nov 9;8:1380. doi: 10.1038/s41467-017-01704-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — AIPR-1 inhibits neuronal Ca²⁺ bursts from the ryanodine receptor. Expression of either aipr-1, ryr-1, or both genes was knocked down in A-type motor neurons using RNAi. a Representative traces of Ca²⁺ transients, determined in A-type motor neurons using GCaMP6f. b Bar graphs of calcium transient frequencies, amplitudes, and area in the wild type (wt) (n = 9), aipr-1 knockdown (n = 10), ryr-1 knockdown (n = 6), and aipr-1 ryr-1 double knockdown (n = 6). Data are shown as mean ± SEM. *p < 0.05, ***p < 0.001 compared with wt; (one-way ANOVA followed by Tukey’s post-hoc test)