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. 2017 Oct 26;8(10):e3139. doi: 10.1038/cddis.2017.552

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Copyright © 2017 The Author(s)

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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Overexpression of HDAC2 promotes CP-treated tubular epithelium cells apoptosis. (a) CP regulated the activity of HDAC2 in HK-2 and mTEC cells. (b) Effects of overexpression of HDAC2 on expression of KIM-1 in HK-2 and mTEC cells analyzed by western blot. (c) Effects of overexpression of HDAC2 on apoptosis in HK-2 and mTEC cells analyzed by flow cytometry. (d) Effects of overexpression of HDAC2 on activities of Caspase-3 in HK-2 and mTEC cells. The activity of caspase-3 was quantified according to the Materials and Methods section. Data were represented as mean±S.D. of three independent experiments. **P<0.01 versus control group, ^&&P<0.01 versus TSA+pEGFP-C1; ^$$P<0.01 versus VPA+pEGFP-C1