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. 2017 Nov 6;8:588. doi: 10.3389/fneur.2017.00588

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Copyright © 2017 Bertoglio, Amhaoul, Van Eetveldt, Houbrechts, Van De Vijver, Ali and Dedeurwaerdere.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Neurodegeneration in KASE WH/CR and KASE SD/H rats. (A) Representative CA1 and CA4/DH images for KASE rats 1 and 12 weeks post-status epilepticus (SE). During both subacute and chronic phases, KASE WH/CR displayed an overall more extensive neurodegeneration than KASE SD/H in the investigated regions (B–F). Kruskal–Wallis test with post hoc Dunn’s test (B–F). *p < 0.05, **p < 0.01. One week post-SE: control WH/CR n = 4–5, KASE WH/CR n = 8–10, control SD/H n = 5 and KASE SD/H n = 6; 12 weeks post-SE: control WH/CR n = 5–6, KASE WH/CR n = 7, control SD/H n = 4–6 and KASE SD/H n = 6. Representative images: CA1 = 40× magnification, CA4/DH = 20× magnification. Scale bar = 50 µm. WH/CR, Wistar Han/Charles River; SD/H, Sprague-Dawley/Harlan; w, week; CA, cornu ammonis; DH, dentate hilus; PC, piriform cortex; KASE, kainic acid-induced status epilepticus.