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. Author manuscript; available in PMC: 2018 Nov 15.
Published in final edited form as: J Immunol. 2017 Oct 9;199(10):3547–3558. doi: 10.4049/jimmunol.1700251

Figure 1. Increased lesion volume and motor deficits in Nlrx1−/− mice following CCI-injury.

Figure 1

A–C) Nissl staining of wild type brains at 3 days post-CCI compared to Nlrx1−/−(D–F) shows increased lesion volume (G) in the absence of NLRX1. (H) Motor deficits are also more prominent in Nlrx1−/− mice compared to wild type at 3–14 days post-CCI. (n=5–7 per group; **p<0.05).