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. 2017 Nov 13;7:15443. doi: 10.1038/s41598-017-15749-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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WBSCR22 gene was over-expressed and predicted a poor clinical outcome in human CRC of the TCGA cohort. (A) The expression of WBSCR22 in 348 clinical CRC specimens in the TCGA cohort. The red bars represented CRC samples having a more than 2-fold higher WBSCR22 expression than normal samples. (B) WBSCR22 levels were compared between normal samples and different NMT stages of CRC samples. A single spot represented the WBSCR22 expression value of an individual sample, and the results were expressed as the mean ± SE. ***p < 0.001. (C) Kaplan-Meier survival curves were plotted according to the different WBSCR22 mRNA level of all CRC patients. p values were obtained from log-rank test, while hazard ratio (HR) and 95% confidence interval (CI) were determined by univariate Cox regression model.