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. 2017 Oct 4;8(51):88870–88881. doi: 10.18632/oncotarget.21488

Figure 4. miR-216a retrains EMT process of GC cells.

Figure 4

(A) MGC-803 cells that were transfected with precursor miR-216a (pre-miR-216a) or scrambled controls (miR-control) were subjected to immunoblotting for E-cadherin, N-cadherin and Vimentin expression. n = three repeats with similar results, *P<0.05 by t test. (B) IF data revealed that miR-216a overexpression enhanced E-cadherin staining while reduced Vimentin staining in MGC-803 cells. (C) SGC-7901 cells that were transfected with miR-216a inhibitors (anti-miR-216a) or scrambled controls (anti-control) were subjected to immunoblotting for E-cadherin, N-cadherin and Vimentin expression. n = three repeats with similar results, *P<0.05 by t test. (D) IF data revealed that miR-216a knockdown reduced E-cadherin staining while enhanced Vimentin staining in SGC-7901 cells.