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. 2017 Apr 18;313(4):E463–E472. doi: 10.1152/ajpendo.00449.2016

Fig. 4.

Fig. 4.

Gestational adaptations in LXR signaling protect against abnormalities in fetoplacental lipid metabolism in mice. A: placenta weight and total cholesterol, triglycerides, and free fatty acids measured in mouse placenta. B: total cholesterol, triglycerides, and free fatty acids measured in fetal hepatic extracts. C: mRNA expression of genes involved in de novo lipogenesis and cholesterol synthesis in fetal liver. D: levels of free fatty acid in maternal serum. E: expression of intracellular lipases in white adipose tissue. F: protein levels of ATGL in white adipose tissue; a.u., arbitrary units. Results are represented as means ± SE (n = 4–8). *P < 0.05, **P < 0.01, ***P < 0.001, comparison of wild-type (WT) vs. LXR DKO group. P values were determined by unpaired t-test.