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. 2017 Aug 7;8(49):85452–85462. doi: 10.18632/oncotarget.20008

Table 3. Associations between the Dietary Inflammatory Index and the risk of gastric cancer (n=1,164).

Dietary Inflammatory Index p-Value for trend p-Value for interaction
Tertile range
T1 T2 T3
n=354 n=375 n=435
Case/Control, n (%)
 388 (33.33%)
/776 (66.67%)
96 (27.12)
/258 (72.88)
116 (30.93)
/259 (69.07)
176 (40.46)
/259 (59.54)
Total (n=1,164)
 Crude model 1.0
(ref)
1.20
(0.87, 1.66)
1.83
(1.35, 2.47)
<0.001
 Multivariable model 1.0
(ref)
1.41
(0.98, 2.03)
1.63
(1.15, 2.29)
0.007
Sex
Men (n=747)
 Crude model 1.0
(ref)
1.11
(0.74, 1.67)
1.42
(0.97, 2.07)
0.059 0.013
 Multivariable model 1.0
(ref)
1.34
(0.83, 2.17)
1.31
(0.84, 2.05)
0.279
Women (n=417)
 Crude model 1.0
(ref)
1.35
(0.81, 2.26)
3.06
(1.82, 5.14)
<0.001
 Multivariable model 1.0
(ref)
1.48
(0.83, 2.62)
2.98
(1.68, 5.30)
<0.001
H. pylori infection
Positive (n=330)
 Crude model 1.0
(ref)
1.24
(0.87, 1.78)
1.65
(1.18, 2.32)
0.003 0.189
 Multivariable model 1.0
(ref)
1.40
(0.95, 2.07)
1.50
(1.04, 2.16)
0.037
Negative (n=834)
 Crude model 1.0
(ref)
1.53
(0.54, 4.36)
3.00
(1.12, 7.99)
0.021
 Multivariable model 1.0
(ref)
1.59
(0.52, 4.84)
2.93
(1.02, 8.43)
0.036

Adjusted for total caloric intake, body mass index, education, smoking (pack-years), ethanol amount, physical activity, H. pylori infection, and first-degree family history of gastric cancer; The tertile ranges of DII were categorized based on the distribution of the control group (T1: <0.96, T2: 0.96-2.97, T3: ≥2.97).