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. 2017 Sep 25;14(6):6463–6470. doi: 10.3892/ol.2017.7045

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Copyright: © Gan et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Effect of HMGN5 depletion on the expression of Akt, p-Akt, slug, E-cadherin, VEGF-C, cytochrome c, cleaved-caspase-3 and cleaved-PARP. After infection with lentivirus as indicated, 5637 cells were treated with CDDP, with or without IGF-1. The expression of Akt, p-Akt, slug, E-cadherin, VEGF-C, cytochrome c, cleaved-caspase-3 and cleaved-PARP was then analyzed by western blotting. β-actin served as a loading control. CDDP, cisplatin; IGF-1, insulin-like growth factor-1; PARP, poly ADP ribose polymerase; VEGF, vascular endothelilal growth factor; HMGN5, high mobility group nucleosome-binding domain 5.