Skip to main content
. Author manuscript; available in PMC: 2017 Nov 20.
Published in final edited form as: Nature. 2017 May 17;545(7655):452–456. doi: 10.1038/nature22367

Extended Data Figure 10. Chromatin states of human PD1hi tumor-infiltrating CD8+ T cells and model for CD8 TST differentiation and dysfunction in tumors.

Extended Data Figure 10

a, Sorting scheme of peripheral blood lymphocytes for Naïve (N), Effector Memory (EM), Central Memory (CM) CD8 T cell populations (left), and PD1hi CD8 TIL from melanoma and NSCLC patients. b, Differentially accessible ATAC-seq peaks grouped by DESeq-defined differential accessibility pattern. Each column represents one biological replicate. Samples shown include CD45RA+ CD45RO- (Naïve; N; grey), CD45RA- CD45RO+, CD62L- (Effector Memory; EM; light green) and CD45RA- CD45RO+, CD62L+ (Central Memory; CM; dark green) peripheral blood CD8+ T cells from healthy donors, and CD45RA- CD45RO+, PD1hi CD8+ T cells isolated and flow-sorted from human melanoma and lung tumors (PD1hi TIL; blue). Open, accessible chromatin regions are presented in red; inaccessible chromatin regions are presented in blue. c, ATAC-seq signal profiles of SELL in N, EM and CM. Blue boxes highlight peaks that remain accessible in CM or become inaccessible in EM compared to N respectively. d, ATAC-seq signal profiles of IFNG, EGR2, CD5, CTLA4. Pink and blue boxes highlight peaks that become accessible or inaccessible in PD1hi TIL compared to N or CM respectively. e, Model for tumor-specific CD8 T cell differentiation and dysfunction in tumors.