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. 2017 Nov 16;14:223. doi: 10.1186/s12974-017-0994-3

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — Comparison of microglial morphology in the SN among different pre-treated groups. The results displayed that intracranial LPS injection caused microglial activation respectively characterized by “amoeboid form” at 7 days (c, d) or “typical activated form” at 14 days (e, f) post to the LPS exposure. However, endotoxic tolerant preconditioning resulted in fewer microglial counting and lighter immunohistochemical staining (d, f, and h). Since the control group and the PBS (striatum) group were not statistically significant at each time point, we selected 7 days as a representative (a, b). Scale bars, 100 μm (100×), 50 μm (200×), and 25 μm (400×)