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. 2017 Nov 17;7:15797. doi: 10.1038/s41598-017-16124-8

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effects of MyD88 inhibition on TNF-α and IL-1β levels 24 h post-SAH. A substantial increase in TNF-α (a) and IL-1β (b) was found in anterior basal temporal lobes from SAH rats 24 h after SAH. MyD88 inhibition successfully reduced SAH-induced increase of TNF-α and IL-1β expression. The upper panel shows representative protein levels of TNF-α and IL-1β. The bottom panels are quantitative data. Tubulin was used as loading control. Data are expressed as mean ± SD (n = 6 in each group). TNF-α, tumor necrosis factor-α, IL-1β, Interleukin-1β, Definition of SAH is the same as in Fig. 2. ***p < 0.001 compared with the sham group, ^###p < 0.001 compared with the SAH group, NS, no statistic difference compared with the SAH group.