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. Author manuscript; available in PMC: 2019 May 1.
Published in final edited form as: Mitochondrial DNA A DNA Mapp Seq Anal. 2017 May 19;29(4):587–593. doi: 10.1080/24701394.2017.1325480

Table 1.

Donor demographics.

Donor Samples
With CAD
Age Sex Mitochondrial
Haplogroup
Cardiac Pathology
Sample 1 75 M HV-Group History of myocardial infarction, CAD diagnosis
Sample 2 66 F L0, L1, L2 Extensive CAD diagnosis, history of myocardial infarction
Sample 3 86 F L0, L1, L2 CAD diagnosis, >50% stenosis noted in multiple coronary arteries
Sample 4 70 M L0, L1, L2 History of myocardial infarction, atherosclerosis, and ischemic cardiomyopathy
Sample 5 19 F T Diagnosed heart disease, increased cardiac enzymes at time off death
Sample 6 42 M UK Myocardial infarct noted on autopsy, hypertensive cardiovascular disease diagnosis
Sample 7 42 M UK Diagnosed severe CAD, up to 60% stenosis noted in multiple coronary arteries.
Sample 8 68 M H Expired due to respiratory failure. Severe coronary artery disease noted.
Donor Samples Without CAD
Sample 9 57 M H Expired due to intracranial bleed. No history of CAD, thromboembolic disease, atherosclerosis was noted.
Sample 10 70 M I Expired due to cancer. No history of CAD, thromboembolic disease, or atherosclerosis noted.
Sample 11 67 F Y Expired due to anoxia. No history of CAD, thromboembolic disease, or atherosclerosis noted.
Sample 12 80 F UK Expired due to hypoxia. No history of CAD, thromboembolic disease, or atherosclerosis noted.
Sample 13 47 F V Expired due to liver failure. No history of CAD, thromboembolic disease, or atherosclerosis noted.
Sample 14 34 F V Expired due to organ failure. No history of CAD, thromboembolic disease, or atherosclerosis noted.
Sample 15 71 M H Expired due to cancer. No history of CAD, thromboembolic disease, or atherosclerosis noted.