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. 2017 Nov 15;8:928. doi: 10.3389/fphys.2017.00928

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Copyright © 2017 Hao, Liu, Pan, Wang, Gao, Fan and Han.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Endothelial function improved by Rhy is Src- and Akt-dependent in SHR renal arteries. (A) Effects of 12 h-incubation with Rhy (30, 100, or 300 μM) or vehicle on Akt phosphorylation in the arteries from SHRs (n = 4). (B) Effects of wortmannin (a PI3K inhibitor, 10 μM) on the Rhy-improved relaxation (n = 6). (C) Effects of wortmannin (a PI3K inhibitor, 10 μM) on the Rhy-increased eNOS phosphorylation in SHR renal arteries (n = 4). (D) Effects of incubation with Rhy (30, 100, or 300 μM) or vehicle for 12 h on Src phosphorylation in the arteries from SHRs (n = 4). (E) Effects of PP2 (a Src inhibitor, 10 μM) on the Rhy-improved endothelium-dependent relaxation in SHR renal arteries (n = 6). (F) Effects of PP2 (a Src inhibitor, 10 μM) on the Rhy-increased eNOS phosphorylation in SHR renal arteries (n = 4). ^*p < 0.05 vs. vehicle; #p < 0.05 vs. Rhy.