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. 2017 Nov 21;7:15929. doi: 10.1038/s41598-017-16175-x

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Overexpression of miR-30e inhibits the ability of cell proliferation, migration and invasion in BC cells. (a) Relative miR-30e expression levels were analyzed by qRT-PCR in MDA-MB-231/ miR-NC and MDA-MB-231/miR-30e stable cell lines. (b) Over-expression of miR-30e arrested cell proliferation in MDA-MB-231 cells. (c) Cells were treated as above. A sterile 20 μL pipette tip was used form a wound gap. The wound gaps were photographed (top) and measured (bottom). Forced expression of miR-30e also markedly reduced the wound-healing rate. (d) MiR-30e over-expression reduced cell invasion in MDA-MB-231 cells. Data represent mean ± SD. of three replicates. *Indicates significant difference at P < 0.05; **indicates significant difference at P < 0.01.