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TGFβ-induced fak gene transcription and role of ERK signaling. Activity of the fak promoter-luciferase construct (pGL2+FAK), normalized to rinella and expressed as percent of the respective non-treated control, is significantly higher in TGFβ–treated cells (P=0.007 vs. non-treated respective controls, n=4 for each) and in cells pretreated with the MEK-specific inhibitor PD98059 (ctr control)
