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. 2017 Nov 28;22:24. doi: 10.1186/s11658-017-0056-y

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© The Author(s) 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — A10 cells were treated with 2 mmol/l of H₂O₂ for 2 h. Superoxide dismutase (SOD) activity and lipid peroxidation were tested to reflect the level of oxidative stress. Overexpression of HSP27 significantly increased SOD activity (a) and decreased the malondialdehyde level (b) in A10 cells infected with LVUT^Hspb1 compared with A10 cells infected with LVUT^EGFP. These two indexes showed no significant difference between non-infected controls and LVUT^EGFP (n = 3)