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. 2017 Dec 5;37(6):BSR20170772. doi: 10.1042/BSR20170772

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© 2017 The Author(s).

This is an open access article published by Portland Press Limited on behalf of the Biochemical Society and distributed under the Creative Commons Attribution License 4.0 (CC BY).

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(A) FA treatment (0–50 μM) dose dependently induced the activation of ERK1/2. (B) FA treatment (20 μM) time dependently induced the activation of ERK1/2. (C) FA treatment (0–50 μM) dose dependently induced the activation of MEK. (D) FA treatment (20 μM) time dependently induced the activation of MEK.