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. 2017 Nov 28;17:158. doi: 10.1186/s12890-017-0490-7

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Effect of Arg1 ablation in the lung on the expression of inflammatory genes. Black bars represent Arg1-Con mice and white bars Arg1-KO^Tie2 mice. mRNA abundance (AU = number of mRNA copies normalized to 18S rRNA expression and multiplied by 10⁴) of the T_H2-related inflammatory genes Il4, Il13, Il5 and Ccl11, the macrophage-chemotactic protein Ccl2, the anti-inflammatory gene Il10, the T_H1-related inflammatory genes Tnfa and Ifng, and the marker genes for the activation of bronchiolar epithelium Muc5ac and Clca3 are shown as means ± SEM (n = 7-8 mice per group). Significance symbols: *: P < 0.05 (OVA/OVA vs. PBS/OVA Arg1-Con); #: P < 0.05 (OVA/OVA vs. PBS/OVA Arg1-KO^Tie2); ‡: P < 0.05 (OVA/OVA Arg1-KO^Tie2 vs OVA/OVA Arg1-Con)