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. 2017 Aug 24;8(55):93567–93579. doi: 10.18632/oncotarget.20510

Table 3. Association between primary tumor sidedness and gene alterations of TK receptors/RAS pathway/PI3K pathway (N = 201).

Primary tumor sidedness P-value
Variable Right (n = 56) Left (n = 145)
ERBB2 status
 Wild-type 54 132 0.243
 Mutanta 2 13
MET status
 Wild-type 55 139 0.676
 Mutanta 1 6
EGFR status
 Wild-type 56 141 0.578
 Mutant 0 4
FGFR1 status
 Wild-type 56 135 0.065
 Mutant 0 10
PDGFRA status
 Wild-type 55 144 0.481
 Mutant a 1 1
KRAS status
 Wild-type 29 97 0.047
 Mutant 27 48
NRAS status
 Wild-type 54 142 0.620
 Mutant 2 3
HRAS status
 Wild-type 55 143 0.999
 Mutant 1 2
BRAF status
 Wild-type 41 136 < 0.001
 Mutant 15 9
MAPK2K1 status
 Wild-type 54 137 0.729
 Mutant 2 8
PTEN status
 Wild-type 39 107 0.554
 Mutant b 17 38
PIK3CA status
 Wild-type 41 127 0.014
 Mutant 15 18
Alterations in TK receptors/RAS pathway/PI3K pathway
 0 6 41 0.024
 1 24 56
 2 or more 26 48
 All wild-type 6 41 0.009
 Mutant-type 50 104

a Including mutation and amplification.

b Including mutation and deletion