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. 2017 Nov 29;8:1850. doi: 10.1038/s41467-017-02020-2

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 10 — Cellular response and controlled self-assembly of Me1P3T. a, b Cellular toxicity of the oligothiophene analogue Me1P3T (green) was first eliminated by complexation with HSA, which subsequently mediates its membrane translocation. c The cell sorts Me1P3T into the mitochondria followed by its transport into the perinuclear region. Self-assembly can be induced or impeded in specific compartments via external stimuli (temperature/inhibitor). In this way, Me1P3T was locked in the mitochondria using bafilomycin and initiated self-assembly by temperature reduction (red)