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. 2017 Nov 30;8:2044. doi: 10.3389/fpls.2017.02044

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Copyright © 2017 Xu, Lei, Li, Zhang, Zhao, Zhao, Fan, Li, Yang, Shang, Xiao and Wang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Mutation, knocking-down and overexpressing of XCT in Col-gl do not change disease phenotypes. (A,B) Representative plants (A) and powdery mildew infected-leaves (B) of the indicated lines show the morphology and disease phenotype, respectively. Note the freshly emerged light-green leaves in xct-5 (arrowheads) in comparison with those in Col-gl and the complemented line xct-5/XCT (arrow). (C) Representative leaves from the indicated lines show the disease phenotype of powdery mildew at 10 dpi. (D) Quantification of sporulation at 10 dpi. Error bars represent SD (n = 6). (E,F) Growth of the virulent strain P. syringae DC3000 (E) and the non-pathogenic mutant strain P. syringae DC3000 (hrcC^-) (F) in the indicated lines. Error bars indicate SD (n = 6). Similar results were obtained in three independent experiments.