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. 2017 Dec 4;216(12):4027–4040. doi: 10.1083/jcb.201612018

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© 2017 Mathew et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms/). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 International license, as described at https://creativecommons.org/licenses/by-nc-sa/4.0/).

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Figure 9. — Model illustrating stress-induced transcriptional changes at RPGs liberating transcriptional regulators such as Sfp1 regulated by TORC1 signaling, Cmr1, and spliceosomes, leading to spliceosome disassembly and splicing defects. Subsequently, Hsh155 is sequestered in INQ and eventually cytoplasmic aggregates, which are regulated by chaperones until stress recovery.