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. 2017 Nov 29;10:56. doi: 10.1186/s13041-017-0334-7

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Overexpression of PKMζ augments the size of large-amplitude miniature excitatory postsynaptic currents (mEPSCs). a The mEPSC frequency of neurons expressing PKMζ K98W, PKMζ T227E, or control neurons at DIV 16–17 (One-way ANOVA, p = 0.1697, n = 20–21 neurons). b The mean mEPSC amplitude of neurons expressing PKMζ K98W, PKMζ T227E, or control neurons at DIV 16–17 (One-way ANOVA, p = 0.1483). c The amplitude of 8 largest mEPSCs from each cell (PKMζ K98W, n = 20 neurons, n = 160 events; Control, n = 20 neurons, n = 160 events; PKMζ T227E, n = 21 neurons, n = 168 events; One-way ANOVA with post-hoc Newman Keuls multiple comparison test (**p < 0.01, ***p < 0.001)). Data are represented as mean ± S.E.M.