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. 2017 Dec 5;12(12):e0188715. doi: 10.1371/journal.pone.0188715

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© 2017 Parker et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 3 — Animal numbers: lynx1WT/α6WT = 7, lynx1KO/α6WT = 7, lynx1WT/α6L9′S = 7, lynx1KO/α6L9′S = 5. Left panel: α-CtxMII-sensitive efflux, mediated via α6β2* nicotinic receptor (total efflux, minus efflux in the presence of α-CtxMII). Lynx1WT/α6WT and lynx1KO/α6WT are significantly different (p < 0.001), designated by ^. Lynx1WT/α6L9′S and lynx1KO/α6L9′S are significantly different (p = 0.005), designated by *. Middle panel: α-CtxMI-Iresistant efflux, mediated by α4β2* nicotinic receptors (efflux in the presence of α-CtxMII). There are no significant differences between lynx1WT/α6WT and lynx1KO/α6WT or between lynx1WT/α6L9′S and lynx1KO/α6L9′S. Right panel: total ⁸⁶Rb⁺ efflux in superior colliculus. Lynx1WT/α6WT and lynx1KO/α6WT are significantly different (p = 0.032), designated by ^. Lynx1WT/α6L9′S and lynx1KO/α6L9′S are significantly different (p = 0.003), designated by *.