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. 2017 Nov 16;12(12):1974–1983. doi: 10.2215/CJN.01280217

Table 2.

Genotype-phenotype correlation of 89 children in the Nephronophthisis Registry carrying mutations in various ciliary genes

Genotype Senior Løken Joubert COACH COMA Jeune/MSS CKD Age at ESRD, yr (1) Liver Involvement Ocular Involvement CNS Involvement Airways Involvement Skeletal Involvement
NPHP1, n=60 7 5 8 98 11.4±2.4 (n=43) 8 41 25 8 2
NPHP3, n=4 100 0.7; 4; 8; 10 (n=4) 100 25 50
NPHP4, n=5 100 14 (n=1) 20
NPHP5/IQCB1, n=5 100 100 14; 16; 16; 27 (n=4) 100 20
NPHP6/CEP290, n=5 100 60 7; 21 (n=2) 100 100 40
NPHP11/TMEM67, n=6 33 33 17 67 3; 6; 8; 18 (n=4) 100 83 83
IFT140, n=4 100 100 7; 10 (n=2) 25 100 25 100

Cell contents are the percentages of participants in a given row (genotype) for which the phenotype listed in the column header was observed or the ages (as means±SD or lists of individual ages) at which ESRD developed. COACH, cerebellar vermis hypoplasia, oligophrenia, ataxia, coloboma, hepatic fibrosis; COMA, congenital oculomotor apraxia; MSS, Mainzer–Saldino syndrome; CNS, central nervous system; —, 0% of patients.