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. 2017 Dec 7;23(45):7989–7999. doi: 10.3748/wjg.v23.i45.7989

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©The Author(s) 2017. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Pathological images of mouse liver injected with 200 protoscolices at different time points. 0 d: On the same day of portal vein injection, the protoscolices congested the portal vein. 1 d: The injected protoscolice caused the dilatation of vessel sinusoids. The central vein size increased. 3 d: Those dead protoscolices ended up as focal degeneration; mouse liver reacted with increased diameter of central veins, inflammatory cell infiltration (blue stained), and collagen deposition (red stained). 1 wk: Mouse liver had protective immune reactions such as lymphocyte infiltration. The vessel fibrosis was found in biliary ducts and portal vein area. 3 wk: The newly developed hydatid cyst was found full of the germinal layer, the laminated layer, and the beginning of the adventitious layer. 6 wk: The mouse localized the hydatid cyst with compressed and fibrotic host tissue (HE staining, × 10).