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. 2017 Aug 16;1:2470547017724744. doi: 10.1177/2470547017724744

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© The Author(s) 2017

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 2. — Effects of UCMS on DNA/RNA oxidation in frontal cortex (FC) and basolateral amygdala (BLA). Representative immunostaining for NeuN (red) and 8-OHdG/8-oxo-G (green) and co-staining (merge) in the FC (a) or BLA (b) of nonstress mice and mice subjected to UCMS. The original magnification was × 20. Scale bar represents 50 µm. Quantitative analysis of the number of NeuN/8-OHdG/8-oxo-G co-labeled cells using Metamorph count nuclei software revealed a significant increase in neuronal DNA/RNA oxidation in the FC (c) and no change in the BLA (d). *p < 0.05 compared to nonstress animal group.