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. 2017 Dec 14;8:2119. doi: 10.1038/s41467-017-02304-7

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — IL-21 or IL-33 treatments induce clearance of established BPS persistence. Plasmids expressing murine IL-21 or IL-33 and empty vector control were injected into BPS HDI mice at 4 and 7 weeks post injection (w.p.i.) (arrows). Sera were collected at indicated time points and levels of IL-21 and IL-33 (a), HBsAg (b), HBsAb (c) and HBeAg (d) were analysed using ELISA. Group means and s.e.m. within group are presented with group sizes (n) indicated. Group positivity percentage data are also presented for HBsAg (b, right panel), HBsAb (c, right panel) and HBeAg (d, right panel). HBV DNA for each group were analysed using pooled serum in commercial quantitative assay (e). Dotted lines represent cut-off thresholds (a, b–d, right panels) and lower limit of quantification (e) respectively. geq genome equivalents