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. 2017 Oct 27;8(60):102078–102087. doi: 10.18632/oncotarget.22158

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Copyright: © 2017 Xu et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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(A) The results of trans-well assay (representative micrographs are presented) demonstrated the inhibition of invasion and migration in the sgRNA-KLF4(3) group. (B) The relative invasion and migration rate was calculated. (C) A wound healing assay indicated that the overexpression of KLF4 in T24 cells resulted in a retardation of wound closure compared with the control group. (D) EMT progression was suppressed in the sgRNA-KLF4(3) group. The data were expressed as the means ± S.D. ^* P < 0.05.