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. 2017 Dec 14;8:2131. doi: 10.3389/fpls.2017.02131

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Copyright © 2017 Malka and Cheng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Schematic representation of TRP-independent and TRP-dependent auxin biosynthesis. IAOx metabolized from TRP acts as key metabolic branching point for biosynthesis of indole GLS, camalexin, and IAA. Inactivation of genes of post-acetaldoxime GLS biosynthesis leads to elevated IAA along with impaired indole GLS. Dashed arrows indicate pathways that are not well defined. INS, INDOLE SYNTHASE; TSA, TRP SYNTHASE A; TSB, TRP SYNTHASE B; TRP, tryptophan; CYP79B2/B3, CYTOCHROME P450 MONOOXYGENASE 79B2/CYTOCHROME P450 MONOOXYGENASE 79B3; SUR2, SUPERROOT2; SUR1, SUPERROOT1; UGT74B1, S-GLUCOSYLTRANSFERASE 74B1; GLS, glucosinolates; IAOx, indole-3-acetaldoxime; TAA1, TRP AMINOTRANSFERASE OF ARABIDOPSIS; IPA, indole-3-pyruvic acid; YUC, YUCCA; TAM, tryptamine; IAD, indole-3-acetaldehyde; IAM, indole-3-acetamide; AMI1, AMIDASE1; IAN, indole-3-acetonitrile; NIT, NITRILASE; CYP71B15, CYTOCHROME P450 MONOOXYGENASE 71B15; IAA, indole-3-acetic acid.