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. 2017 Dec 19;7:17775. doi: 10.1038/s41598-017-18195-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Bar charts of fold-change data. (a) 24 major immune mediators, of which 21 were significantly dysregulated in patients with acute EBV infection (AIM) from both our primary cohort (Shanghai) and the Minnesota cohort. Additional 3 important immune mediators (IFNG, IL6, and TNF) were also included in the chart although their expression levels were just borderline different in either of the two cohorts. The “significantly different” means adjusted p < 0.05; and the “borderline different” indicates an adjusted p-value of 0.05 to 0.20. The panel b shows the fold-change data for 26 T cell receptors (TCRs), and the panel c for 5 killer cell lectin-like receptors (KLRs), 4 killer cell immunoglobulin-like receptors (KIRs), and 2 other receptors and 2 transcription factors that are important for differentiation of lymphocytes.