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. 2017 Dec 20;18:234. doi: 10.1186/s13059-017-1362-4

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Analysis of published data identifies markers of ontogeny. a The intersection of: (1) genes that are differentially expressed between blood-derived and microglial TAMs in mouse (outer circle); (2) their homologues; (3) genes expressed (mean CPM > 1) in human BMDM/microglia from non-malignant tissue; and (4) TAMs from human gliomas (n = 16 patients). b Distributions of the log2 ratios (human BMDMs over microglia) (y-axis) for the differentially expressed murine-TAM homologues from (a)