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. 2017 Dec 20;4(4):ofx231. doi: 10.1093/ofid/ofx231

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© The Author(s) 2017. Published by Oxford University Press on behalf of Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 6. — Hepatitis B virus (HBV)–specific CD8+ T-cell responses are decreased in a patient developing chronic HBV infection. Peripheral blood mononuclear cells (PBMCs) were isolated from an HBV genotype A–infected patient who evolved to chronic infection after acute HBV infection. These PBMCs were stimulated with HBV-derived peptides and TNFα, IL-2, MIP-1β, and IFNγ production was measured via flowcytometry. Peptide pools: C, core protein; E, envelope protein; PI, polymerase protein pool I; PII, polymerase protein pool II; X, x protein. Abbreviations: GT, genotype; HBV, hepatitis B virus; n.a., not available.