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. 2017 Dec 8;9(34):1261–1269. doi: 10.4254/wjh.v9.i34.1261

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©The Author(s) 2017. Published by Baishideng Publishing Group Inc. All rights reserved.

Open-Access: This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/

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Morphological changes in liver before and after ischemia and reperfusion, transmission electron micrographs of representative images of liver sections from one patient. A: Baseline, before induction of ischemia, shows the normal state of liver morphology at a cellular level; B: Morphology of a sinusoid with neighbouring hepatocytes 20 min after ischemia; C: Representative image of sinusoid and haptocytes 20 min after reperfusion; D: Endothelial lining of a sinusoid with hepatocyte microvilli; E: Morphology of the Space of Disse post-ischemia; F: Morphology of the space of disse post-reperfusion. SD: Space of disse; H: Hepatocyte; EC: Endothelial cells; VL: Vessel lumen; K: Kupffer cell. Arrow shows the absence of hepatocyte microvilli.