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. 2017 Dec 19;8:921. doi: 10.3389/fphar.2017.00921

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Copyright © 2017 Silva-Cruz, Carlström, Ribeiro and Sebastião.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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LTP induced by strong-θ-burst is inhibited when CB₁Rs are continuously activated. (A) Time course of the averaged fEPSP slopes, and original traces of fEPSP recordings, in control conditions (no drugs) or in the presence of 500 nM WIN (CB₁R agonist), or 500 nM WIN together with 1 μM AM251. (B) Quantification of LTP magnitude under the indicated drug conditions. Data in control conditions in panels (A) and (B) are the same as shown in Figures 2A,B, but is represented in this figure to allow comparison with the drug conditions. ^∗∗∗∗p < 0.0001 (F_(2,33) = 10.3, one-way ANOVA with Sidak’s correction). For further details see legend to Figure 1.