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. 2017 Dec 19;11:358. doi: 10.3389/fncel.2017.00358

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Copyright © 2017 Liu, Liu, Chao, Liu, Liu, Zheng, Chen, Gong, Teng, Li, Wang and Xue.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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EMAP-II combined with anti-miR-330-3p and PKCα activator enhance the effects of DOX on inhibiting the cell viabilities and promoting the apoptosis of U87 glioma cells. (A) The cell viabilities of U87 glioma cells were assessed by MTT. (B) Apoptosis analysis inU87 glioma cells was evaluated by Annexin V-PE/7-AAD staining. Values are means ± SD (n = 5, each). *P < 0.05, **P < 0.01 vs. control group; ^#P < 0.05 vs. DOX group.