Fig. 5. Effects of caudal fourth ventricular compound C (Cc) administration on hindbrain A2 nerve cell 5′-monophosphate-activated protein kinase (AMPK), phosphorylated AMPK (pAMPK), and dopamine-β-hydroxylase (DβH) protein responses to 12 hours of food deprivation (FD-12) or 18 hours (FD-18) in groups of 30 (E-30) or 300 (E-300) rats. (A) It depicts representative immunoblots of A2 AMPK (row A), pAMPK (row B), DβH (row C), and α-tubulin (row D). (B, C, D) These respectively illustrate mean optical density (O.D.) measures±SEM for A2 AMPK, pAMPK, and DβH protein profiles in groups of E-30 and E-300 animals exposed to FD-12 (left side of each graph) or FD-18 (right side of each graph) with or without Cc treatment. FF, full-fed; V, vehicle. ^aP<0.05 vs. FD-V.